BLOOD COAGULATION IMMEDIATELY AFTER DEATH THE BLOOD IN THE HEART AND IN VEINS STARTS TO COAGULATE (THROUGH THE CHANGE OF FIBRINOGEN TO FIBRIN) RED OR WHITE BLOOD COAGULATES EMERGE, WHICH UNADHERE TO THE WALL RED COAGULATES (CRUOR) ARE FROM THE WHOLE COAGULATED BLOOD ALSO FROM THE RED CELLS BETWEEN THE FIBRES 2x THEY ARE DARK RED, MOIST, SOFTLY ELASTIC WHITE COAGULATES EMERGE BY SLOW COAGULATION WHEN ERYTHROCYTES HAVE TIME TO SEDIMENT THEY DON’T CONTAIN ERYTHROCYTES WHEN THEY CONTAIN MORE PLASMA THEY ARE YELLOW TRANSPARENT AND MORE MOIST (AMBER COAGULATES) 4x COAGULATION...COAGULATION...COAGULATION...OF BLOOD MICROSCOPICALLY, THE POSTMORTAL COAGULATES USUALLY DO NOT CONTAIN COAGULATINES OF RED BLOOD CELLS... 4x COAGULATION...COAGULATION...COAGULATION...OF BLOOD ...AND BY THAT, THE DIFFER FROM INTRAVITAL BLOOD COAGULATES (TROMBS) MICROSCOPICALL, TROMBS ARE MORE DRY, HARD, FRIABLE USUALLY ADHERENT TO THE WALL OF THE VEIN OR OF THE HEART THE COAGULATION SPEED DEPENDS ON THE VOLUME OF CO2 IN BLOOD BLOOD WHICH IS RICH ON CO2 DOES NOT HAVE TO COAGULATE - IT STAYS FLUID: - DEATH BY HEAVY ASPHYXIATION - POISONING BY H2S AND CO2 ALSO BY TETANUS A CONSEQUENCE OF HYPERTHERMIA OR DEATH BY ELECTRICAL FLOW (DURING POSTMORTAL ACIDOSIS, BECAUSE OF CO2 OVERLOAD THE FIBRINOGEN AUTOLYTHICALLY DISSECTS BEFORE ITS OWN COAGULATION A YELLOW ATROPHY OF LIVER - PROTHROMBINE ABSENTS IN THE BLOOD IT DOES NOT COAGULATE ANYMORE, BECAUSE THE COAGULATES LATER BECOME FLUID AGAIN THROUGH THE AUTOLYSIS OF FIBRINE AND THE WORK OF ROT BACTERIAE) (JULY 2006)